New molecular mechanism in diastolic heart failure.

نویسندگان

  • Arnold M Katz
  • Michael R Zile
چکیده

In contrast to systolic heart failure (SHF), for which knowledge of pathophysiology and therapy has advanced rapidly over the past decade, little is known about diastolic heart failure (DHF). The article by van Heerebeek et al1 in this issue of Circulation that describes an abnormal distribution of titin isoforms in DHF may herald a new approach to understanding the pathophysiology of this syndrome.

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عنوان ژورنال:
  • Circulation

دوره 113 16  شماره 

صفحات  -

تاریخ انتشار 2006